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Ly-6chi monocytes dominate hypercholesterolemia-associated monocytosis and give rise to macrophages in atheromata. Monocyte subsets differentially employ ccr2, ccr5, and cx3cr1 to accumulate within atherosclerotic plaques. Identification of splenic reservoir monocytes and their deployment to inflammatory sites.

Distinct functions of chemokine receptor axes in the atherogenic mobilization and recruitment of classical monocytes. Ly-6chigh monocytes depend on nr4a1 to balance both inflammatory and reparative phases in the infarcted myocardium. Rosenfeld ME, Ross R. Macrophage and smooth muscle cell proliferation in atherosclerotic lesions of whhl and comparably hypercholesterolemic fat-fed rabbits. Local proliferation dominates lesional macrophage accumulation in atherosclerosis.

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Increased expression of matrix metalloproteinases and matrix degrading activity in vulnerable regions of human atherosclerotic plaques. Consequences and therapeutic implications of macrophage apoptosis in atherosclerosis: The importance of lesion stage and phagocytic efficiency.

Morphological and biomechanical aspects of vulnerable coronary plaque. Archives des maladies du coeur et des vaisseaux. The role of macrophages and dendritic cells in the clearance of apoptotic cells in advanced atherosclerosis. European journal of immunology. Phagocyte receptors for apoptotic cells: Recognition, uptake, and consequences. Programmed necrosis: Backup to and competitor with apoptosis in the immune system.

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Ann Biomed Eng. Circ Cardiovasc Imaging. Spotty calcification typifies the culprit plaque in patients with acute myocardial infarction: An intravascular ultrasound study. Non-invasive anatomic and functional imaging of vascular inflammation and unstable plaque. Compensatory enlargement of human atherosclerotic coronary arteries. Remodeling of coronary arteries in human and nonhuman primates.

Computed tomographic angiography characteristics of atherosclerotic plaques subsequently resulting in acute coronary syndrome. J Am Coll Cardiol. Lysosomal cysteine proteases in atherosclerosis. Genetic inactivation of il-1 signaling enhances atherosclerotic plaque instability and reduces outward vessel remodeling in advanced atherosclerosis in mice. Libby P, Theroux P. Pathophysiology of coronary artery disease. Induction of collagenase, stromelysin, and tissue factor.

Cd40 ligation induces tissue factor expression in human vascular smooth muscle cells. Edgington TS. Surfing tissue factor. From the predicted to the discovery and elucidation of unanticipated functions and biology of potential significance. Thromb Haemost. Gimbrone MA, Jr. Vascular endothelium, hemodynamics, and the pathobiology of atherosclerosis. Cardiovasc Pathol. Il6r genetics consortium emerging risk factors collaboration. Interleukin-6 receptor pathways in coronary heart disease: A collaborative meta-analysis of 82 studies.

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Low-dose colchicine for secondary prevention of cardiovascular disease. Ridker PM. Targeting inflammatory pathways for the treatment of cardiovascular disease. Progress and challenges in translating the biology of atherosclerosis. Resolution of inflammation: State of the art, definitions and terms.

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Diminished lipoxin biosynthesis in severe asthma. American journal of respiratory and critical care medicine. Impaired phagocytosis in localized aggressive periodontitis: Rescue by resolvin e1. PLoS One. Aspirin-triggered lipoxin and resolvin e1 modulate vascular smooth muscle phenotype and correlate with peripheral atherosclerosis.

Circulating and dietary omega-3 and omega-6 polyunsaturated fatty acids and incidence of cvd in the multi-ethnic study of atherosclerosis. Journal of the American Heart Association. Wu JH, Mozaffarian D. Omega-3 fatty acids, atherosclerosis progression and cardiovascular outcomes in recent trials: New pieces in a complex puzzle. Combined therapy of dietary fish oil and stearoyl-coa desaturase 1 inhibition prevents the metabolic syndrome and atherosclerosis.

Omega-3 fatty acids ameliorate atherosclerosis by favorably altering monocyte subsets and limiting monocyte recruitment to aortic lesions. Dietary n-3 lcpufa from fish oil but not alpha-linolenic acid-derived lcpufa confers atheroprotection in mice. J Lipid Res.

Orally administered eicosapentaenoic acid induces rapid regression of atherosclerosis via modulating the phenotype of dendritic cells in ldl receptor-deficient mice. Lack of ep4 receptors on bone marrow-derived cells enhances inflammation in atherosclerotic lesions. Cardiovasc Res.

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Cell Metab. Infection regulates pro-resolving mediators that lower antibiotic requirements. D-series resolvin attenuates vascular smooth muscle cell activation and neointimal hyperplasia following vascular injury. Low-dose aspirin for the prevention of atherothrombosis. J Exp Med.

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Current understanding of the role of b cell subsets and intimal and adventitial b cells in atherosclerosis. Adaptive immunity in atherosclerosis: Mechanisms and future therapeutic targets. Nat Rev Cardiol. Subendothelial lipoprotein retention as the initiating process in atherosclerosis: Update and therapeutic implications.

Macrophages in atherosclerosis: A dynamic balance. Identification of a danger-associated peptide from apolipoprotein b apobds-1 that triggers innate proatherogenic responses. Nlrp3 inflammasomes are required for atherogenesis and activated by cholesterol crystals. Am Heart J. Cd36 coordinates nlrp3 inflammasome activation by facilitating intracellular nucleation of soluble ligands into particulate ligands in sterile inflammation.

Lack of toll-like receptor 4 or myeloid differentiation factor 88 reduces atherosclerosis and alters plaque phenotype in mice deficient in apolipoprotein e. Modulation of atherosclerosis in mice by toll-like receptor 2. Cd36 ligands promote sterile inflammation through assembly of a toll-like receptor 4 and 6 heterodimer. Toll-like receptor 4 deficiency decreases atherosclerosis but does not protect against inflammation in obese low-density lipoprotein receptor-deficient mice.

The critical role of il-1 receptor-associated kinase 4-mediated nf-kappab activation in modified low-density lipoprotein-induced inflammatory gene expression and atherosclerosis. Genetic ablation of irak4 kinase activity inhibits vascular lesion formation. Biochem Biophys Res Commun. Deficient cdtraf6 signaling in leukocytes prevents atherosclerosis by skewing the immune response toward an antiinflammatory profile. The lps2 mutation in trif is atheroprotective in hyperlipidemic low density lipoprotein receptor knockout mice.

Innate immunity. Reduced atherosclerosis in mydnull mice links elevated serum cholesterol levels to activation of innate immunity signaling pathways. Macrophages generate reactive oxygen species in response to minimally oxidized low-density lipoprotein: Toll-like receptor 4- and spleen tyrosine kinase-dependent activation of nadph oxidase 2. Vaccines modulating lipoprotein autoimmunity as a possible future therapy for cardiovascular disease. J Intern Med. Gordon S, Martinez FO.

Alternative activation of macrophages: Mechanism and functions. Molecular endocrinology. Exploring the full spectrum of macrophage activation. Reversal of hyperlipidemia with a genetic switch favorably affects the content and inflammatory state of macrophages in atherosclerotic plaques. Hdl promotes rapid atherosclerosis regression in mice and alters inflammatory properties of plaque monocyte-derived cells.

Antagonism of mir in mice promotes reverse cholesterol transport and regression of atherosclerosis. Effects of native and myeloperoxidase-modified apolipoprotein a-i on reverse cholesterol transport and atherosclerosis in mice. Rapid regression of atherosclerosis: Insights from the clinical and experimental literature. Nat Clin Pract Cardiovasc Med.

Infusion of reconstituted high-density lipoprotein leads to acute changes in human atherosclerotic plaque. Interleukin protects from atherosclerosis and modulates plaque composition by skewing the macrophage phenotype. Reprogramming macrophages to an anti-inflammatory phenotype by helminth antigens reduces murine atherosclerosis. Gerrity RG. The role of the monocyte in atherogenesis: Ii. Migration of foam cells from atherosclerotic lesions.

Studies of hypercholesterolemia in the nonhuman primate. Changes that lead to fatty streak formation. Emigration of monocyte-derived cells from atherosclerotic lesions characterizes regressive, but not progressive, plaques. The neuroimmune guidance cue netrin-1 promotes atherosclerosis by inhibiting the emigration of macrophages from plaques. Regression of atherosclerosis is characterized by broad changes in the plaque macrophage transcriptome. Gene expression changes in foam cells and the role of chemokine receptor ccr7 during atherosclerosis regression in apoe-deficient mice.

Statins promote the regression of atherosclerosis via activation of the ccr7-dependent emigration pathway in macrophages. Neuroimmune guidance cue semaphorin 3e is expressed in atherosclerotic plaques and regulates macrophage retention. Therapeutic sirna silencing in inflammatory monocytes in mice.

Nat Biotechnol. Effects of atorvastatin on early recurrent ischemic events in acute coronary syndromes: The miracl study: A randomized controlled trial. Risk of thrombosis in human atherosclerotic plaques: Role of extracellular lipid, macrophage, and smooth muscle cell content.

Heart J. Multislice computed tomographic characteristics of coronary lesions in acute coronary syndromes. Spotty calcification as a marker of accelerated progression of coronary atherosclerosis: Insights from serial intravascular ultrasound. Activation of lipoxin a 4 receptors by aspirin-triggered lipoxins and select peptides evokes ligand-specific responses in inflammation. Novel functional sets of lipid-derived mediators with antiinflammatory actions generated from omega-3 fatty acids via cyclooxygenase 2-nonsteroidal antiinflammatory drugs and transcellular processing.

Resolvins: A family of bioactive products of omega-3 fatty acid transformation circuits initiated by aspirin treatment that counter proinflammation signals. Novel docosatrienes and 17s-resolvins generated from docosahexaenoic acid in murine brain, human blood, and glial cells. Autacoids in anti-inflammation.

J Biol Chem. Maresins: Novel macrophage mediators with potent antiinflammatory and proresolving actions. Resolvin d2 is a potent regulator of leukocytes and controls microbial sepsis. Novel proresolving aspirin-triggered dha pathway. Resolvin d3 and aspirin-triggered resolvin d3 are potent immunoresolvents.

Mobilizing lipocortin 1 in adherent human leukocytes downregulates their transmigration. Resolvin d series and protectin d1 mitigate acute kidney injury. Interleukin 10 il inhibits cytokine synthesis by human monocytes: An autoregulatory role of il produced by monocytes. Journal of Experimental Medicine. Resolvin e1 and protectin d1 activate inflammation-resolution programmes. Anti-inflammatory role of the murine formyl-peptide receptor 2: Ligand-specific effects on leukocyte responses and experimental inflammation.

Resolvin d1 binds human phagocytes with evidence for proresolving receptors. Resolvin e2 formation and impact in inflammation resolution. Macrophage phagocytosis of apoptotic neutrophils is critically regulated by the opposing actions of pro-inflammatory and anti-inflammatory agents: Key role for tnfalpha.

Macrophage proresolving mediator maresin 1 stimulates tissue regeneration and controls pain. Resolvin d1 controls inflammation initiated by glutathione-lipid conjugates formed during oxidative stress. Br J Pharmacol. Resolvin d1 and its precursor docosahexaenoic acid promote resolution of adipose tissue inflammation by eliciting macrophage polarization toward an m2-like phenotype. Resolvin d1-mediated nox2 inactivation rescues macrophages undertaking efferocytosis from oxidative stress-induced apoptosis.

Biochem Pharmacol. Bazan NG. Neuroprotectin d1 npd1 : A dha-derived mediator that protects brain and retina against cell injury-induced oxidative stress. Brain pathology. Aspirin-triggered lipoxin a4 blocks reactive oxygen species generation in endothelial cells: A novel antioxidative mechanism. Annexin peptide ac suppresses tnfalpha-induced inflammatory responses via inhibition of rac1-dependent nadph oxidase in human endothelial cells.

Resolvin e1 regulates inflammation at the cellular and tissue level and restores tissue homeostasis in vivo. For that reason, we believe we should recognize and reward those men and women on the front lines and support positions. We believe when we take care of the team members, they will take care of the customer and when the customer is happy, the money will follow. We are looking forward to great things from this new partnership.

With over professionals located throughout North America, the companies help business owners release the wealth of their business by providing merger, acquisition, and wealth management services. View source version on businesswire. Carl Doerksen cdoerksen generational. Instead of its typical path through Manhattan, this year's parade was kept to the area in front of Macy's flagship store and aimed at a television audience instead of live crowds.

More than 25 million Americans reported not having enough food to eat in the past week. Mexican migrants deported from the United States celebrated Thanksgiving on Thursday with a sense of nostalgia for the country where they went in search of a new life. The Macy's Thanksgiving Day Parade marches on, but the balloons are distancing.

Counter-terrorism experts believed it could be linked to the attacks last year on London Bridge and in Streatham which could have inspired an uptick in extremism and surge in activity by law enforcement and other agencies.

There were a total of 6, referrals to Prevent between April and March - up 10 per cent from a record low of 5, the previous year. The number of Prevent referrals for concerns over right-wing extremism dropped slightly in the latest year, to 1, from 1, in the 12 months to March It came as Social media firms were warned they face legally-enforced codes to prevent and remove terrorist and child abuse content online under new duty of care laws to be announced next month.

Speaking at an online Rusi event, Mr Brokenshire said the explosion in social media in the past decade posed new challenges to law enforcement and intelligence agencies. Through the introduction of new legislation, we will establish a legal duty of care on companies towards their users overseen by an independent regulator. The Duke and Duchess of Sussex celebrated their first Thanksgiving as a family in the US on Thursday, sitting down to homegrown vegetables from the garden.

It has been a tumultuous year since the Sussexes left the UK last November to spend their first Thanksgiving as a family-of-three in Canada. They published a Happy Thanksgiving message on their now defunct SussexRoyal Instagram account and later released a photograph, taken that day, of a beaming Duke holding Archie at the waterfront home they had rented on Vancouver Island. Iowa farmers grew the state's first legal hemp crop on about acres.

The pandemic means no in-person attendance at the 94th annual event. The World Health Organization released guidelines on physical activity Wednesday, urging people to ward off sedentary lifestyles. Donald Trump is expected to issue a wave of pardons, including some related to the Russia investigation, in his final weeks in office. It raised the possibility that the US president may also attempt to pre-emptively pardon himself to avoid any potential future legal entanglements. On Wednesday night Mr Trump pardoned Michael Flynn, his former national security adviser, who had pleaded guilty to lying to the FBI about his conversations with a Russian ambassador.

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